Etiology Self Assessment

Normal Pulp
 TrueFalse
Odontoblasts are found only in the coronal pulp
The lifespan of odontoblasts is 1 - 3 weeks
Fibroblasts are responsible for secondary dentine production
Secondary dentine is more porous than primary dentine
Odontoblastic processes extend to the dentine-enamel border
Stem cells are found in the pulp
Cell free zone of Weil is next to the odontoblastic layer
Cell free zone of Weil is located between odontoblasts and predentine
Dendritic cells form tertiary dentine
Dendritic cells detect and present antigens to other immune cells
Dendritic cells are more densely organised in the periphery than in the central pulp.
Leukocytes are not found in normal pulp.
Pulpal inflammation starts only after the bacteria enter the pulp
Microbial irritants are more important in pulpitis than chemical irritants
Chemical iritation can stimulate secondary dentine formation
Leaking fillings challenge pulp mostly by chemical irritants.
Pulp tolerates better high than low temperatures
Crown preparation without perforation can cause internal bleeding in the pulp
Internal bleeding can cause discoloration of the crown
A necrotic tooth is always infected
Apical periodontitis follows necrosis always within 1 - 3 weeks
Antigens alone cannot initiate inflammation of the pulp
Eugenol can stimulate secondary dentine formation through dentine
Apical periodontitis always follows soon after ischemic necrosis
In acute apical periodontitis (AAP) the lesion may be poorly seen during the first days
Lateral periodontitis is as common as apical periodontitis
The etiology of a lateral lesion is different from that of apical lesion
Osteoclastic cells are involved in bone resorption in apical periodontitis
Cytocines are involved in pulpitis but not in apical periodontitis
Macrophages are not present in the apical lesions
Anaerobic bacteria dominate in primary apical periontitis
Enterococcus faecalis is the most frequent finding in promary apical periodontitis
Enterococcus faecalis is present in ca 50% of retreatment cases
Enteric bacteria are more common in retreatment than in primary apical periodontitis
Fusobacterium nucleatum is often found in spreading infections
Yeasts are typically present in primary apical periodontitis
Fusobacterium nucleatum and Porphyromonas gingivalis are rarely found in abscesses
Caries lesion is the most typical pathway for bacteria to enter the pulp
Bacteria can enter the pulp through a lateral canal
Bacteria cannot enter the pulp through an invagination
Epithelial cells are found in ca 50% of radicular cysts
Bay cysts may respond better for conservative endodontic treatment than true cysts
A fibrous capsule typically surrounds epithelium in a radicular cyst
Bone resorption is always a pathological condition
Bone turn-over rate is ca. 2 - 3 years
Osteoclasts use acidity and hydrolytic enzymes to resrob bone
Pulp is partly vital in active internal inflammatory root resorption
(Root) Surface resorption is rare
Surface resorption cannot heal once started
Surface resorption cannot usually be seen in the radiographs
Reparative cementum can be thicker than normal root surface cementum
Cervical resorption starts coronally to junctional epithelium
A red discoloration (pink spot) may be an indication of cervical resorption
Cervical resorption always perforates the pulp
Type I cervical resorption is the largest type.
Type IV cervical resorption can grow both coronally and apically
Osteoid (hard) tissue can sometimes be detected in cervical resorption lesion inside the tooth
The etiology of multiple cervical resorption is not known
External inflammatory root resorption typically starts after intrusion injury
External inflammatory root resorption proceeds slowly
External inflammatory root resorption involves small areas on the root surface