True | False |
Odontoblasts are found only in the coronal pulp |
The lifespan of odontoblasts is 1 - 3 weeks |
Fibroblasts are responsible for secondary dentine production |
Secondary dentine is more porous than primary dentine |
Odontoblastic processes extend to the dentine-enamel border |
Stem cells are found in the pulp |
Cell free zone of Weil is next to the odontoblastic layer |
Cell free zone of Weil is located between odontoblasts and predentine |
Dendritic cells form tertiary dentine |
Dendritic cells detect and present antigens to other immune cells |
Dendritic cells are more densely organised in the periphery than in the central pulp. |
Leukocytes are not found in normal pulp. |
Pulpal inflammation starts only after the bacteria enter the pulp |
Microbial irritants are more important in pulpitis than chemical irritants |
Chemical iritation can stimulate secondary dentine formation |
Leaking fillings challenge pulp mostly by chemical irritants. |
Pulp tolerates better high than low temperatures |
Crown preparation without perforation can cause internal bleeding in the pulp |
Internal bleeding can cause discoloration of the crown |
A necrotic tooth is always infected |
Apical periodontitis follows necrosis always within 1 - 3 weeks |
Antigens alone cannot initiate inflammation of the pulp |
Eugenol can stimulate secondary dentine formation through dentine |
Apical periodontitis always follows soon after ischemic necrosis |
In acute apical periodontitis (AAP) the lesion may be poorly seen during the first days |
Lateral periodontitis is as common as apical periodontitis |
The etiology of a lateral lesion is different from that of apical lesion |
Osteoclastic cells are involved in bone resorption in apical periodontitis |
Cytocines are involved in pulpitis but not in apical periodontitis |
Macrophages are not present in the apical lesions |
Anaerobic bacteria dominate in primary apical periontitis |
Enterococcus faecalis is the most frequent finding in promary apical periodontitis |
Enterococcus faecalis is present in ca 50% of retreatment cases |
Enteric bacteria are more common in retreatment than in primary apical periodontitis |
Fusobacterium nucleatum is often found in spreading infections |
Yeasts are typically present in primary apical periodontitis |
Fusobacterium nucleatum and Porphyromonas gingivalis are rarely found in abscesses |
Caries lesion is the most typical pathway for bacteria to enter the pulp |
Bacteria can enter the pulp through a lateral canal |
Bacteria cannot enter the pulp through an invagination |
Epithelial cells are found in ca 50% of radicular cysts |
Bay cysts may respond better for conservative endodontic treatment than true cysts |
A fibrous capsule typically surrounds epithelium in a radicular cyst |
Bone resorption is always a pathological condition |
Bone turn-over rate is ca. 2 - 3 years |
Osteoclasts use acidity and hydrolytic enzymes to resrob bone |
Pulp is partly vital in active internal inflammatory root resorption |
(Root) Surface resorption is rare |
Surface resorption cannot heal once started |
Surface resorption cannot usually be seen in the radiographs |
Reparative cementum can be thicker than normal root surface cementum |
Cervical resorption starts coronally to junctional epithelium |
A red discoloration (pink spot) may be an indication of cervical resorption |
Cervical resorption always perforates the pulp |
Type I cervical resorption is the largest type. |
Type IV cervical resorption can grow both coronally and apically |
Osteoid (hard) tissue can sometimes be detected in cervical resorption lesion inside the tooth |
The etiology of multiple cervical resorption is not known |
External inflammatory root resorption typically starts after intrusion injury |
External inflammatory root resorption proceeds slowly |
External inflammatory root resorption involves small areas on the root surface |